More scientists are looking for ways to prolong life, most often they discover that a small change can bring immense benefits.

The latest discovery?
It has been studied that due to inhibition of a particular receptor of pain, the same acting in older people, the lives of laboratory mice lengthens by about 15 percent.
If the results are confirmed in humans, considering an average age of about 80 years, you might enjoy half a decade and more than life.
A few weeks ago scientists said that in mice by increasing a growth factor could repair tissues of the muscles and the brain, reversing the effects of aging.
Quest 'latest study, published in Cell by researchers at the University of Berkeley, looks like something that will be able to completely block one of the causes of aging.
When you feel pain, the nerves in our body signal the brain to stop doing any action that we're hurting-and it is also true that the pain itself might kill us.
The mice that were genetically inhibited the TRPV1 pain receptors showed "a youthful metabolic profile in old age", giving researchers a clue as to why the pain-especially the elderly-may prove to be fatal.
"Older people say they feel pain more young people-and they blame age," said Andrew Dillon, the author of the research.
"We have turned the talk: maybe they are old because they suffer.
Perhaps it is the pain that leads to aging. "
Certainly aging is caused by more than one factor, but the way in which people experience pain may be a common factor, said Dillin.
Often, with age, people in developing the so-called CGRP molecule, which binds to the TRPV1 receptors.
People in which it develops are more likely to suffer from metabolic disorders in the future, which are the cause of many signs of the aging process.
"Try pain probably has negative effects on life span, affects the relationship with food.
If you feel bad you're not hungry and do not eat, "he said.
"If you have no pain you are more inclined to eat and regulate metabolism."
As I said, blocking this receptor genetically pain, the metabolism of older rats returned typical of young mice, making them live better and longer.
Of course, the pain has an important role in the evolution of man; It keeps us away from what we would kill quickly, like putting your hands on the stove on or walk on sharp objects.
The point, as Dillin said, is figuring out how to block the TRPV1 receptor in humans without making too powerful the effect.
There are already migraine medications that attack just this receptor, but are all too effective to promote their use-often those who use them end up not hearing anything and burn yourself, or go into hypothermia.
The inhibitor of TRPV1 would not be prescribed to anyone.
According Dillin should monitor the levels of CGRP molecule to figure out who would have the most benefits.
While in all likelihood completely TRPV1 receptor will never inhibit a good way to extend the average life of people, to develop a system that no extreme effects controls could prove useful.
The receptor is "completely conserved in humans," in the words of Dillin, so there is no reason why the tests on guinea pigs should not have the same results in humans-it remains only to understand how to control their effects.

From Vice